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3.1Incretin Physiology, Glucose & Insulin Regulation GLP-1, GIP and glucagon receptor biology and the enteroinsular axis Glucose-dependent insulin secretion, glucagon suppression and gastric emptying Central appetite and reward signaling studied in the incretin literature Insulin resistance, hyperinsulinemia and the metabolic phenotype in front of you Interpretation of fasting insulin, HOMA-IR, HbA1c, continuous glucose data and lipid subfractions Peptide-adjacent metabolic agents studied alongside incretins (MOTS-c, metformin, 5-Amino-1MQ) Metaflammation: the inflammatory-metabolic overlap that connects this module to Module 6 3.2The GLP-1 / GIP / Glucagon Agent Class in Full Semaglutide: formulations, route options and the STEP and SELECT trial programs Tirzepatide: dual GIP/GLP-1 agonism and the SURMOUNT and SURPASS programs Liraglutide, dulaglutide, exenatide and lixisenatide — class positioning and dosing structures Retatrutide and the investigational triple-agonist pipeline
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There are people who also report improved recovery and lower post-workout muscle soreness
