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At invasion fronts, CAAs release IL-6, TNF-α, leptin, CCL2, and CCL5, recruiting monocytes and biasing TAMs toward M2-like states via STAT3 and ERK/MEK signaling, with downstream VEGF-A production and angiogenesis in breast cancer models

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5-Amino-1MQ is studied primarily as an NNMT inhibitor in laboratory models examining nicotinamide metabolism, cellular metabolic signaling, and related biochemical pathways