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First, the main site of TMA production from both carnitine and γBB were mainly in the bacterial rich cecum, with formation of TMA from γBB occurring at a rate approximately 1.5 fold higher than that from an equimolar amount of L -carnitine (Figure 3A)

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Both work through separate biological mechanisms: semaglutide acts on GLP-1 receptors to reduce appetite and improve glucose control, while L-carnitine supports energy substrate utilization at the cellular level